Attenuation of antimalarial agent hydroxychloroquine on TNF-α-induced endothelial inflammation.
Identifieur interne : 000C50 ( Main/Exploration ); précédent : 000C49; suivant : 000C51Attenuation of antimalarial agent hydroxychloroquine on TNF-α-induced endothelial inflammation.
Auteurs : Ruiru Li [République populaire de Chine] ; Haobo Lin [République populaire de Chine] ; Yujin Ye [République populaire de Chine] ; Youjun Xiao [République populaire de Chine] ; Siqi Xu [République populaire de Chine] ; Jingnan Wang [République populaire de Chine] ; Cuicui Wang [République populaire de Chine] ; Yaoyao Zou [République populaire de Chine] ; Maohua Shi [République populaire de Chine] ; Liuqin Liang [République populaire de Chine] ; Hanshi Xu [République populaire de Chine]Source :
- International immunopharmacology [ 1878-1705 ] ; 2018.
Descripteurs français
- KwdFr :
- Adhérence cellulaire (), Anti-inflammatoires (pharmacologie), Antipaludiques (pharmacologie), Cellules cultivées, Cellules endothéliales de la veine ombilicale humaine (), Cellules endothéliales de la veine ombilicale humaine (métabolisme), Facteur de nécrose tumorale alpha (pharmacologie), Humains, Hydroxychloroquine (pharmacologie), Leucocytes (), Leucocytes (métabolisme), Molécule-1 d'adhérence des cellules vasculaires (métabolisme), Molécule-1 d'adhérence intercellulaire (métabolisme), Système de signalisation des MAP kinases ().
- MESH :
- métabolisme : Cellules endothéliales de la veine ombilicale humaine, Leucocytes, Molécule-1 d'adhérence des cellules vasculaires, Molécule-1 d'adhérence intercellulaire.
- pharmacologie : Anti-inflammatoires, Antipaludiques, Facteur de nécrose tumorale alpha, Hydroxychloroquine.
- Adhérence cellulaire, Cellules cultivées, Cellules endothéliales de la veine ombilicale humaine, Humains, Leucocytes, Système de signalisation des MAP kinases.
English descriptors
- KwdEn :
- Anti-Inflammatory Agents (pharmacology), Antimalarials (pharmacology), Cell Adhesion (drug effects), Cells, Cultured, Human Umbilical Vein Endothelial Cells (drug effects), Human Umbilical Vein Endothelial Cells (metabolism), Humans, Hydroxychloroquine (pharmacology), Intercellular Adhesion Molecule-1 (metabolism), Leukocytes (drug effects), Leukocytes (metabolism), MAP Kinase Signaling System (drug effects), Tumor Necrosis Factor-alpha (pharmacology), Vascular Cell Adhesion Molecule-1 (metabolism).
- MESH :
- chemical , metabolism : Intercellular Adhesion Molecule-1, Vascular Cell Adhesion Molecule-1.
- chemical , pharmacology : Anti-Inflammatory Agents, Antimalarials, Hydroxychloroquine, Tumor Necrosis Factor-alpha.
- drug effects : Cell Adhesion, Human Umbilical Vein Endothelial Cells, Leukocytes, MAP Kinase Signaling System.
- metabolism : Human Umbilical Vein Endothelial Cells, Leukocytes.
- Cells, Cultured, Humans.
Abstract
Hydroxychloroquine (HCQ) is an antimalarial drug that is widely used in the treatment of some autoimmune diseases. In the present study, we explore the role of HCQ in regulating endothelial inflammation and its underlying mechanism.
DOI: 10.1016/j.intimp.2018.08.008
PubMed: 30121047
Affiliations:
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Le document en format XML
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First Affiliated Hospital, Sun Yat-sen University, Guangzhou, Guangdong, PR China.</nlm:affiliation><country xml:lang="fr">République populaire de Chine</country><wicri:regionArea>Department of Rheumatology and Clinical Immunology, The First Affiliated Hospital, Sun Yat-sen University, Guangzhou, Guangdong</wicri:regionArea><wicri:noRegion>Guangdong</wicri:noRegion></affiliation></author><author><name sortKey="Shi, Maohua" sort="Shi, Maohua" uniqKey="Shi M" first="Maohua" last="Shi">Maohua Shi</name><affiliation wicri:level="1"><nlm:affiliation>Department of Rheumatology and Clinical Immunology, The First Affiliated Hospital, Sun Yat-sen University, Guangzhou, Guangdong, PR China.</nlm:affiliation><country xml:lang="fr">République populaire de Chine</country><wicri:regionArea>Department of Rheumatology and Clinical Immunology, The First Affiliated Hospital, Sun Yat-sen University, Guangzhou, Guangdong</wicri:regionArea><wicri:noRegion>Guangdong</wicri:noRegion></affiliation></author><author><name sortKey="Liang, Liuqin" sort="Liang, Liuqin" uniqKey="Liang L" first="Liuqin" last="Liang">Liuqin Liang</name><affiliation wicri:level="1"><nlm:affiliation>Department of Rheumatology and Clinical Immunology, The First Affiliated Hospital, Sun Yat-sen University, Guangzhou, Guangdong, PR China.</nlm:affiliation><country xml:lang="fr">République populaire de Chine</country><wicri:regionArea>Department of Rheumatology and Clinical Immunology, The First Affiliated Hospital, Sun Yat-sen University, Guangzhou, Guangdong</wicri:regionArea><wicri:noRegion>Guangdong</wicri:noRegion></affiliation></author><author><name sortKey="Xu, Hanshi" sort="Xu, Hanshi" uniqKey="Xu H" first="Hanshi" last="Xu">Hanshi Xu</name><affiliation wicri:level="1"><nlm:affiliation>Department of Rheumatology and Clinical Immunology, The First Affiliated Hospital, Sun Yat-sen University, Guangzhou, Guangdong, PR China. Electronic address: xuhanshi@mail.sysu.edu.cn.</nlm:affiliation><country xml:lang="fr">République populaire de Chine</country><wicri:regionArea>Department of Rheumatology and Clinical Immunology, The First Affiliated Hospital, Sun Yat-sen University, Guangzhou, Guangdong</wicri:regionArea><wicri:noRegion>Guangdong</wicri:noRegion></affiliation></author></analytic><series><title level="j">International immunopharmacology</title><idno type="eISSN">1878-1705</idno><imprint><date when="2018" type="published">2018</date></imprint></series></biblStruct></sourceDesc></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Anti-Inflammatory Agents (pharmacology)</term><term>Antimalarials (pharmacology)</term><term>Cell Adhesion (drug effects)</term><term>Cells, Cultured</term><term>Human Umbilical Vein Endothelial Cells (drug effects)</term><term>Human Umbilical Vein Endothelial Cells (metabolism)</term><term>Humans</term><term>Hydroxychloroquine (pharmacology)</term><term>Intercellular Adhesion Molecule-1 (metabolism)</term><term>Leukocytes (drug effects)</term><term>Leukocytes (metabolism)</term><term>MAP Kinase Signaling System (drug effects)</term><term>Tumor Necrosis Factor-alpha (pharmacology)</term><term>Vascular Cell Adhesion Molecule-1 (metabolism)</term></keywords><keywords scheme="KwdFr" xml:lang="fr"><term>Adhérence cellulaire ()</term><term>Anti-inflammatoires (pharmacologie)</term><term>Antipaludiques (pharmacologie)</term><term>Cellules cultivées</term><term>Cellules endothéliales de la veine ombilicale humaine ()</term><term>Cellules endothéliales de la veine ombilicale humaine (métabolisme)</term><term>Facteur de nécrose tumorale alpha (pharmacologie)</term><term>Humains</term><term>Hydroxychloroquine (pharmacologie)</term><term>Leucocytes ()</term><term>Leucocytes (métabolisme)</term><term>Molécule-1 d'adhérence des cellules vasculaires (métabolisme)</term><term>Molécule-1 d'adhérence intercellulaire (métabolisme)</term><term>Système de signalisation des MAP kinases ()</term></keywords><keywords scheme="MESH" type="chemical" qualifier="metabolism" xml:lang="en"><term>Intercellular Adhesion Molecule-1</term><term>Vascular Cell Adhesion Molecule-1</term></keywords><keywords scheme="MESH" type="chemical" qualifier="pharmacology" xml:lang="en"><term>Anti-Inflammatory Agents</term><term>Antimalarials</term><term>Hydroxychloroquine</term><term>Tumor Necrosis Factor-alpha</term></keywords><keywords scheme="MESH" qualifier="drug effects" xml:lang="en"><term>Cell Adhesion</term><term>Human Umbilical Vein Endothelial Cells</term><term>Leukocytes</term><term>MAP Kinase Signaling System</term></keywords><keywords scheme="MESH" qualifier="metabolism" xml:lang="en"><term>Human Umbilical Vein Endothelial Cells</term><term>Leukocytes</term></keywords><keywords scheme="MESH" qualifier="métabolisme" xml:lang="fr"><term>Cellules endothéliales de la veine ombilicale humaine</term><term>Leucocytes</term><term>Molécule-1 d'adhérence des cellules vasculaires</term><term>Molécule-1 d'adhérence intercellulaire</term></keywords><keywords scheme="MESH" qualifier="pharmacologie" xml:lang="fr"><term>Anti-inflammatoires</term><term>Antipaludiques</term><term>Facteur de nécrose tumorale alpha</term><term>Hydroxychloroquine</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Cells, Cultured</term><term>Humans</term></keywords><keywords scheme="MESH" xml:lang="fr"><term>Adhérence cellulaire</term><term>Cellules cultivées</term><term>Cellules endothéliales de la veine ombilicale humaine</term><term>Humains</term><term>Leucocytes</term><term>Système de signalisation des MAP kinases</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Hydroxychloroquine (HCQ) is an antimalarial drug that is widely used in the treatment of some autoimmune diseases. In the present study, we explore the role of HCQ in regulating endothelial inflammation and its underlying mechanism.</div></front></TEI><affiliations><list><country><li>République populaire de Chine</li></country><region><li>Guangdong</li></region><settlement><li>Jiangmen</li></settlement></list><tree><country name="République populaire de Chine"><noRegion><name sortKey="Li, Ruiru" sort="Li, Ruiru" uniqKey="Li R" first="Ruiru" last="Li">Ruiru Li</name></noRegion><name sortKey="Liang, Liuqin" sort="Liang, Liuqin" uniqKey="Liang L" first="Liuqin" last="Liang">Liuqin Liang</name><name sortKey="Lin, Haobo" sort="Lin, Haobo" uniqKey="Lin H" first="Haobo" last="Lin">Haobo Lin</name><name sortKey="Shi, Maohua" sort="Shi, Maohua" uniqKey="Shi M" first="Maohua" last="Shi">Maohua Shi</name><name sortKey="Wang, Cuicui" sort="Wang, Cuicui" uniqKey="Wang C" first="Cuicui" last="Wang">Cuicui Wang</name><name sortKey="Wang, Jingnan" sort="Wang, Jingnan" uniqKey="Wang J" first="Jingnan" last="Wang">Jingnan Wang</name><name sortKey="Xiao, Youjun" sort="Xiao, Youjun" uniqKey="Xiao Y" first="Youjun" last="Xiao">Youjun Xiao</name><name sortKey="Xu, Hanshi" sort="Xu, Hanshi" uniqKey="Xu H" first="Hanshi" last="Xu">Hanshi Xu</name><name sortKey="Xu, Siqi" sort="Xu, Siqi" uniqKey="Xu S" first="Siqi" last="Xu">Siqi Xu</name><name sortKey="Ye, Yujin" sort="Ye, Yujin" uniqKey="Ye Y" first="Yujin" last="Ye">Yujin Ye</name><name sortKey="Zou, Yaoyao" sort="Zou, Yaoyao" uniqKey="Zou Y" first="Yaoyao" last="Zou">Yaoyao Zou</name></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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